研究论文
唐学玺,李永祺.久效磷对海洋微藻毒性机理的初步研究Ⅲ.超氧化物歧化酶和过氧化物酶活性的变化[J].环境科学学报,1998,18(2):204-207
久效磷对海洋微藻毒性机理的初步研究Ⅲ.超氧化物歧化酶和过氧化物酶活性的变化
- A PRELIMINARY STUDY ON TOXICITY MECHANISM OF MONOCROTOPHOS ON MARINE MICROALGAE Ⅲ.CHANGES OF ACTIVITY OF SUPEROXIDE DISMUTASE AND PEROXIDASE
- 摘要：报道了久效磷对3种海洋微藻细胞内2种清除活性氧的关键性酶---超氧化物歧化酶和过氧化物酶活性的影响。结果显示：1.在久效磷的胁迫下,扁藻和三角褐指藻细胞的超氧化物歧化酶(SOD)活性均表现出下降的总变化趋势,而叉鞭金藻细胞的SOD活性时而上升,时而下降,在整个胁迫过程中呈现出无规律性的变化。2.随着久效磷胁迫时间的延长,3种微藻细胞的过氧化物酶活性均逐渐下降,表现出相同的变化规律性。这说明不同的藻种,久效磷对其细胞内酶活性的影响不尽相同。推测超氧化物歧化酶和过氧化物酶(POD)活性的降低是微藻细胞内过量产生活性氧,进而引起藻细胞膜脂过氧化伤害的主要原因之一。
- Abstract：This paper reports the effect of monocrotophos stress on the activity of superoxide dismutase (SOD) and peroxidase (POD) which are two key enzymes to eliminate active oxygen in algal cell. Results showed that: 1. SOD activity on Platymonas sp. and Phaeodactylum tricornutum decreased under moncrotophos stress, and fluctuated irregularly in Dicrateria sp. during the whole period of stress; 2. POD activity of three marine microalgae decreased gradually with the prolonging time of stress. Therefore, the mechanism to eliminate active oxygen is different in various marine microalgae. It is one of the main causes for activity decline of SOD and POD to lead to the excess production of active oxygen and cell damage of lipid peroxidation.
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